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Zero2Cool
2020-08-17T16:12:21Z
Originally Posted by: KRK 

I thought this was an interesting read.
https://www.medicalnewst...-weather-affect-covid-19 



Horrible read.

Quote:

If COVID-19 is seasonal, experts will likely establish this in 2021 or 2022 after the main pandemic waves.



Don't want this BS to continue any further.
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KRK
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2020-08-17T16:23:14Z
Originally Posted by: Zero2Cool 

Horrible read.



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beast
2020-08-17T18:37:22Z
Originally Posted by: KRK 

Nevertheless, as mentioned in an earlier post, there wasn't blind study when penicillin was prescribed in WWII...that decisions saved thousands of lives. HCQ is far safer than penicillin especially at low doses

There also wasn't credit cards, power steering, super glue, McDonald's franchise or single computer modem when penicillin was prescribed in WWII. This ain't 1940...

And you keep arguing the safety of HCQ, but H2O is even safer than HCQ and Penicillin but absolutely nothing I've seen proves that any of them stops covid-19 in humans, as none of them stop TMPRSS2 enzyme which carries the Covid-19 into lung cells.

HCQ became popular thought to stop Covid-19 after a research study on monkeys showed HCQ stops Covid-19 in monkeys... but their bodies use a different enzyme, which HCQ does stop.

If humans cells used the same enzyme as monkeys, HCQ would have been a great to stop Covid-19. But it doesn't.

Now, some studies suggest Covid-19 isn't killing people, but the human body's immune system reaction to Covid-19 is killing people. In pure wild ass speculation, I wonder if HCQ prevents the immune system reaction in certain ways, which makes the symptoms looks at they've improved. Which would explain both why some doctors with patients swear by it, and why the science say it doesn't prevent Covid-19.

But the scientific studies have seemed to suggest HCQ doesn't stop Covid-19 from entering the cells.

Originally Posted by: KRK 

Point taken....and a fair observation...but I am just a pissed at Trump for listening to him for WAAAAY to long. Somewhere over the last 3 years, his balls fell off.

I try to avoid politics, especially online as it's often pointless pissing contests,

But Trump still has major balls, as he's always had more balls than brains. Then again he's in good company, as lack of brains seems to be a running tread with Presidents. Sadly it's a popularity contest not a who's the best for the job contest.

Originally Posted by: KRK 

No, actually it accentuates their favorable results with HCQ. Their systems are vastly inferior, but their death rates are much lower per case. I am suggesting HCQ has alot to do with that.

Favorite results with HCQ, favoritable results with Mask, favoritable results with mostly free and universal health care (except Indonesia, in which John Hopkins suggests your graph was extremely wrong in terms of Indonesia).

Also those results completely hid the results of countries that disagree with it, such as Iran, Brazil, Japan, South Korea.

So that information not only cherry picks the countries of the effect to match it's desired results, but it also cherry picks the cause it wants to push to claim what got that desired results.


Originally Posted by: KRK 

I am tired of other people, with questionable motivations, dictating policy, especially when those decisions may be costing 10s of thousands of live.

You seem to just tired because the person in this case doesn't agree with you... it seems as if the person did agree with you, you might be all for it, but then again that's politics.

Originally Posted by: KRK 

If we can agree that patients and Dr. should be permitted to make the choice, I am fine.

If something isn't going to be that harmful and than absolutely.

Though I absolutely think there should be certain limits where we don't have doctor's doing completely stupid shit (not talking about HCQ here) or that have a much higher risk of doing harm than helping (again not talking about HCQ here).

But even myself personally, when I was a child I had problem that caused pain and led to 8 different surgeries (doctor and Parents of patients approved). The surgeries temporarily helped, but also caused a different long term harm, and I believe the harm has been worse than the helpfulness.

Come to find, today, some Hospitals have decided to refuse to do such a surgery because they've deemed it more harmful than helpful, and as someone who's lived through it, I'd agree with that decision... even if the doctor and patient agreed because (at least in my particular case, I can't speak for others, nor what the average is, which would be extremely important) the harm clearly out weighed the benefit.

So I think there does need to be a check and balances where the doctor can't just come up with any idea, as something that might help one thing, can seriously hurt another.

But as for HCQ, I'd certainly be alright with more people trying it, especially with a study, seeing if it clears up the over-aggressive immune reactions.

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Zero2Cool
2020-08-17T18:49:26Z
Basically, what we have here is experts upon experts not coming to the same conclusion. And yet, we have people making decisions based on the inconclusive findings. Not small decisions either, major decisions. Aaron Rodgers was right when he spoke about the ill effects no fans in Lambeau is going to have on Green Bay. We fucking need the fans in the seats watching the Packers! We need the fans flocking here in droves -- buying things. You selfish fucks, let the boomer remover do it's thang!!

Okay, only quarter/half serious about that very last part. Green Bay seriously does need the fans. The stadium holds something like 80 thousand and we have a population of like 105 thousand. I promise you all those fans filling the seats aren't from the immediate area.

When reading these studies and the numbers and how they've come to their conclusion, or lack there of. I can't help but thinking of that one dentist who refused to endorse the toothpaste their four colleagues did. Did that one dentist know more than the others? Did that one dentist do more thoroughly testing? Did that one dentist refuse payment? I mean, what the hell! Who can you trust?

The answer is ... NO ONE.

What the experts need to do is inform us. They need to educate us. That's what we should all be asking for and demanding nothing less, nothing more. Inform me of the risks. Inform me of the measures to minimize/eliminate the risk. When (not if) I screw up suffer the consequences the risk; adequately provide that information on how to best eradicate it.
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2020-08-17T18:59:09Z
Beast benevolently but begrudgingly bequeathed
Quote:

But as for HCQ, I'd certainly be alright with more people trying it

That's all I can ask.

Yeah Baby Yeah 
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beast
2020-08-17T19:14:06Z
Originally Posted by: Zero2Cool 

What the experts need to do is inform us. They need to educate us. That's what we should all be asking for and demanding nothing less, nothing more. Inform me of the risks. Inform me of the measures to minimize/eliminate the risk. When (not if) I screw up suffer the consequences the risk; adequately provide that information on how to best eradicate it.



Absolutely! Experts, ie not politicians...

Though, I agree, two problems still exist

1) What about the items that there are no current eradicate methods available.

2) Payment, I believe our current for profit system is only set up to help pay for pre-approved items for certain items. And encourage only to treat what's currently happening, not preventing future items.

This is both why people can't get stuff no on an already approved list and why I can't get an easy and painless screening to see if I have the same thing as one of my parents despite having some of the early signs of it. It'd be nice to know for sure.
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2020-08-18T14:54:36Z
This is a paper re face shields...I have long felt that this thing is just as likely to get into the eyes as the nose.
Quote:

https://jamanetwork.com/journals/jama/fullarticle/2769693?guestAccessKey=9a4c2b12-5d3c-4a7f-a78c-77651b07cb0a&utm_source=silverchair&utm_medium=email&utm_campaign=article_alert-jama&utm_content=olf&utm_term=081720
Results
Before face shields, 62 workers (40 women) visited 5880 homes with 31 164 persons. From the 5880 homes visited, 222 persons tested positive for SARS-CoV-2, between May 4 to May 13. Twelve workers (19%) were infected during this period. Eight developed symptoms (fever, cough, sore throat, myalgia, and anosmia) and 4 were asymptomatic. The 12 infected workers were moved to care centers. Four developed desaturation and mild breathing difficulty and were treated with oral hydroxychloroquine and oxygen therapy; all 4 recovered. Contact tracing of the workers who tested positive identified 14 van drivers, who were monitored. All were asymptomatic and tested negative between day 7 and 10 after contact with the workers.

After face shields, 50 workers (previously uninfected) continued to provide counseling, visiting 18 228 homes. Among the counseled, 118 428 persons, 2682 subsequently tested positive for SARS-CoV-2. No worker developed asymptomatic or symptomatic infection.



If you don't want to wear a face shield, and I don't, wearing a hat and glasses can't hurt.
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Zero2Cool
2020-08-18T15:33:26Z
Originally Posted by: KRK 

This is a paper re face shields...I have long felt that this thing is just as likely to get into the eyes as the nose.

If you don't want to wear a face shield, and I don't, wearing a hat and glasses can't hurt.



What kind of hat? What kind of glasses?

The face shields have already been ruled out for kids in our school district. It has to be a face covering or mask.
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KRK
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2020-08-18T15:42:32Z
Keep it simple. Wear a Packers baseball hat with any glasses....prescription or otherwise, just to keep droplets out of the eyes.
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Zero2Cool
2020-08-18T16:12:28Z
Originally Posted by: KRK 

Keep it simple. Wear a Packers baseball hat with any glasses....prescription or otherwise, just to keep droplets out of the eyes.



We went from being told only those who have it should wear a mask, to city ordinates requiring masks to the state mandating masks. I'm thinking the next step is this. I'm going to order a couple and deck them out with Packers gear!

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2020-08-20T10:43:30Z
Kind of interesting
Quote:

Sniffing Out Where Virus First Strikes in COVID-19
— More context for why COVID-19 patients might lose sense of smell
by Molly Walker, Associate Editor, MedPage Today August 19, 2020

https://www.medpagetoday...HE_dual-gmail-definition 

When examining how SARS-CoV-2 enters the body, one part of the nose may provide further insights into transmission, symptoms, and potential avenues of treatment for COVID-19, researchers suggested from a lab study of human tissue samples.

ACE2 receptor expression was 200- to 700-fold greater in the olfactory epithelium -- the center of smell detection -- relative to other areas of the nose and trachea, reported Andrew Lane, MD, of Johns Hopkins Outpatient Center in Baltimore, and colleagues. ACE2 is the primary portal through which the SARS-CoV-2 virus enters host cells.

These findings "suggest that active virus infection and replication occurs in the apical layer of nasal and olfactory mucosa," they wrote in the European Respiratory Journal.

"The olfactory epithelium is quite an easy part of the body for a virus to reach, it's not buried away deep in our body, and the very high levels of ACE2 that we found there might explain why it's so easy to catch COVID-19," co-author Mengfei Chen, PhD, also of Johns Hopkins, said in a statement.

The researchers noted prior research suggesting a link between ACE2 expression level and COVID-19 associated clinical traits, such as increased severity in young patients with obesity "linked to increased ACE2 expression in lung epithelial cells."

In addition, they highlighted one of the more unusual symptoms, and sometimes the only presenting symptom of COVID-19: anosmia, but without "concomitant nasal inflammatory symptoms."

"The loss of the sense of smell suggests the possibility of direct targeting by SARS-CoV-2 of the olfactory system," they wrote. "However, the cellular location of ACE2 protein in the olfactory epithelium has not been previously demonstrated."

Lane and colleagues examined tissue samples from the back of the nose of 23 patients (19 with chronic sinusitis, the others healthy controls) removed during endoscopic procedures, and biopsies from the trachea of seven patients. None of the participants had tested positive for COVID-19 or were suspected of infection; all were older than 30.

ACE2 was not detected on olfactory neurons, however.

Tobias Welte, MD, of Hannover University School of Medicine in Germany, who was not involved with the research, characterized this as a "clever study" that examines why COVID-19 patients may have loss of smell as their only symptom.

"It suggests that the part of our nose responsible for smelling could also be the place where the coronavirus gains a foothold in the body," said Welte, past president of the European Respiratory Society, in a statement. "This finding will need to be confirmed, but it offers possible new avenues for treating the infection."

Lane and colleagues noted that the differential expression of ACE2 in the "olfactory neuroepithelium and respiratory epithelium" could not only account for the spectrum of nasal-related symptoms, but could provide some insights into novel treatments.

"Whether nasal saline irrigation, a common treatment for sinonasal conditions, is beneficial or counterproductive in SARS-CoV-2 infection remains to be determined; however, consideration should be given to the delivery of topical anti-viral additives, such as detergent or povidone-iodine, directed at the nasal and nasopharyngeal viral reservoirs," they wrote.

Molly Walker is an associate editor, who covers infectious diseases for MedPage Today. She has a passion for evidence, data and public health. Follow

Euorpean Respiratory Journal Source Reference: Chen M, et al "Elevated ACE2 expression in the olfactory neuroepithelium: implications for anosmia and upper respiratory SARS-CoV-2 entry and replication" Eur Respir J 2020; DOI: 10.1183/13993003.01948-2020.


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Cheesey
2020-08-20T16:30:27Z
How many lawyer commercials have you seen recently telling you about drugs or other products that were tested and deemed safe, to now “causing cancer” or some other malady like “anal leakage”? (Just kidding about that last one).
So we put our trust in scientists and doctors, only to find down the road that what they said was safe, ended up making people sick or killing them?
It will take years, maybe decades before we will know if any cure they come up with might end up killing more than curing?
Like Zero said, “4 out of 5 dentists recommend it” might mean the 5th one might have decided to not take the bribe to endorse their product.
The old “more hospitals use such and such aspirin then other brands” SOUNDS like that means their brand is better, when chances are it’s the one that sells it cheaper to the hospitals then other brands can, thus more hospitals use their brand.
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KRK
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2020-08-21T01:19:38Z
Lock-downs don't work...Peru had an onerous 4 month lock-down.

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2020-08-21T13:44:23Z
I thought this was very interesting....links to Sunlight study and T-Cell study are in the article. Sunlight is also important for Vitamin D.
Quote:

T-Cell Immunity Underestimated in Predictions for COVID-19 Herd Immunity: Study

BY MEILING LEE August 20, 2020 Updated: August 20, 2020

https://www.theepochtime...noe&utm_medium=email 

There has been a lot of discussion on herd immunity to COVID-19 lately as new studies suggest that people’s immune system has the ability to recognize the virus, explaining why certain individuals have only mild symptoms or none at all.
Herd immunity happens when a large number of people are immune to a disease—either from prior infection or vaccination—which acts to stop or slow down the spread, thus protecting the entire community, including those who have not had the illness.

Estimates for percentages needed for herd immunity has mainly focused on the role of antibodies of those who have recovered from COVID-19, although scientists are still unsure how long the antibodies protect against reinfection.

Studies are now also beginning to consider the contribution of T-cells to herd immunity for COVID-19.

T-Cell Immunity for COVID-19

COVID-19 Antibody Testing Collection Event at the Miami International Mall in Doral, Fla., on June 18, 2020. (Jason Koerner/Getty Images for BioReference Laboratories)

Director of the National Institutes of Health Dr. Francis Collins said in his blog post that studies on the immune response to SARS-CoV-2, the virus that causes the COVID-19 disease, have concentrated mostly on the body’s production of antibodies to determine if we will have immunity against the disease.

“But, in fact, immune cells known as memory T-cells also play an important role in the ability of our immune system to protect us against many viral infections, including—it now appears—COVID-19,” Collins said.

T-cells (along with B-cells) are a type of white blood cell that plays an essential part in the adaptive immune system by assisting the body in destroying and remembering antigens—foreign substances that invade the body. Although T-cells are produced in the bone marrow, they travel to the thymus, a small organ between the lungs directly behind the sternum in the chest, where they will mature into different types of T-cells with specific roles.

As people get older, the thymus shrinks, affecting the production and functionality of T-cells, and so adults 60 and older become more susceptible to infections. The elderly population has been the most affected in the pandemic, where at least 45 percent “of all COVID deaths are coming from 0.62 percent of the population—those in nursing homes and assisted living facilities,” Dr. Tom Reed, a double board-certified medical and surgical specialist, told a Facebook discussion hosted by Texas state Sen. Bob Hall.

A study published in Nature that Collins cites in his blog suggests that the immune system’s T-cells may offer protection against COVID-19 by “remembering past encounters with other human coronaviruses,” explaining why certain people can fight off the illness or experience only mild symptoms.

There are six coronaviruses besides SARS-CoV-2 that infect humans—four of them cause the common cold, while two caused the 2003 Severe Acute Respiratory Syndrome (SARS) outbreak and the 2012 Middle East Respiratory Syndrome (MERS) outbreak. These coronaviruses produced both memory T-cells and antibodies in infected individuals, but it was the memory T-cells that continued to confer immunity for over a decade, according to the study.

Multiple studies have been published since the Nature study confirming similar results.

The latest one to be published in Cell (pdf) shows a more promising role for T-cells in developing herd immunity against COVID-19.

Researchers found that “donors exhibited robust memory T-cell responses months after infection, even in the absence of detectable circulating antibodies specific for SARS-CoV-2, indicating a previously unanticipated degree of population-level immunity against COVID-19.”

Dr. Scott Atlas, President Donald Trump’s new advisor on the pandemic, said last month that emerging data showed that people who didn’t get infected have been found to have COVID-19 immunity. “This is probably due to this T-cell immunity, which is present, and is now shown to last for years,” he said.

Atlas, speaking during an appearance on Fox News, pointed to research from Singapore and from Sweden’s Karolinska Institute. Researchers from the institute stated that many people with mild or asymptomatic cases of COVID-19 demonstrate T-cell-mediated immunity to SARS-CoV-2.

Achieving Herd Immunity

It is generally thought that an estimated 60 to 70 percent of the population needs to become infected by SARS-CoV-2 before herd immunity is achieved. “However, several recent studies have suggested that depending on specific population factors (population density, propensity for travel, susceptibility, etc.), this number may be considerably lower in some sub-populations in particular places around the country,” Dr. Michael Devine, dual-board certified internist and geriatrician and co-founder of Devine Concierge Medicine, told The Epoch Times in an email.

But when asked if some states are already seeing herd immunity, Devine says he doesn’t believe that is the case just yet.
“In every measure we do have at present, the perceived proportion of the population who is believed to have acquired (by having been infected directly) or innate (naturally occurring or genetically predisposed) immunity falls vastly short of any model prediction that would infer herd immunity,” Devine said.

“The reduction in cases currently being enjoyed in some states is the byproduct of healthy population habits—social distancing, utilization of face coverings when in public, and maintaining good hand hygiene.”

Devine also said that limiting travel, the warmer weather, and people “spending more time outdoors, rather than being cooped up in close quarters” may be contributing to the lessening prevalence of the virus.

In a live Instagram session on Aug. 13, Dr. Anthony Fauci, the director of the National Institute of Allergy and Infectious Diseases, said that sunlight kills the virus. “That’s one of the reasons why outside, in the sun, when you are interacting that, that is much better than being inside.”

Another important reason for people to be outdoors is to allow their skin to be exposed to direct sunlight for 15-30 minutes without being sunburnt, to activate the production of vitamin D—an important hormone that helps to fight off infections and regulate the immune system. Studies have found that vitamin D deficiency is associated with increased COVID-19 infection risk.

A study by the Georgetown University Medical Center in 2016 found that sunlight, specifically low-level blue light, also “energizes T cells that play a central role in human immunity” by making them move faster.

“T cells, whether they are helper or killer, need to move to do their work, which is to get to the site of an infection and orchestrate a response,” Gerard Ahern, PhD and lead researcher of the study said in a press release. “This study shows that sunlight directly activates key immune cells by increasing their movement.”

Areas of Possible Herd Immunity

States like Oregon, Washington, and Illinois—where large crowds of people are protesting and rioting frequently without social distancing and where some are not wearing masks—are not seeing a significant spike in new cases or deaths. The Washington Department of Health tweeted on Aug. 14 that the state’s “COVID-19 cases are plateauing” even in King County where the riots continue to take place.

Oregon reported its lowest daily COVID-19 cases on Aug. 17 of 189 and no new deaths, bringing the number of total cases to 23,451 and a death toll of 388.

In Illinois, while daily cases began to rise again towards the end of June with the increasing number of tests given, the fatality rates had begun to significantly decline at the same time.

And in Ventura County in California, a church protesting Gov. Gavin Newsom’s lockdown mandate has been offering indoor services for its congregants (of up to several thousand people) for the past two months without any virus outbreaks, according to NTD. While the church does focus on properly sanitizing before and after services, Pastor Rob McCoy of Godspeak Calvary Chapel leaves it up to church members if they want to wear masks and physical distancing. The pastor and his church members are facing legal consequences for attending an in-person church service after breaking a temporary restraining order to not hold indoor activities.

One possible explanation for lower deaths in these communities is that natural herd immunity has taken effect to some degree.

Zachary Steiber contributed to this story.


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2020-08-23T18:47:52Z
More on T-cells and Immunity...
Quote:

Even mild COVID-19 may produce long lasting immunity

A new study suggests that T cells might provide individuals who had a mild or asymptomatic case of COVID-19 with lasting immunity against future infection, even if their blood contains no neutralizing antibodies.

Research from Sweden suggests that people with mild or asymptomatic cases of COVID-19 may have immunity from future infection.

https://www.medicalnewst...ce-long-lasting-immunity 

Most people who are exposed to SARS-CoV-2, the coronavirus that causes COVID-19, only experience mild symptoms or none at all.

However, the infection can still pass from them to other people, and the overall case fatality rate appears to be converging on 0.5–1.0%.

It is, therefore, important to establish whether individuals who have contracted the virus once can contract it again and become contagious, or whether they are immune to future infection.

“In the absence of a protective vaccine, it is critical to determine if exposed or infected people, especially those with asymptomatic or very mild forms of the disease who likely act inadvertently as the major transmitters, develop robust adaptive immune responses against SARS-CoV-2,” says Marcus Buggert, an immunologist at the Karolinska Institutet in Solna, Sweden.

Research suggests that not all individuals who have contracted SARS-CoV-2 in the past produce antibodies capable of neutralizing the virus, particularly if they only experienced a mild infection.

Studies have also found that immune cells known as memory B cells, which produce antibodies against previously encountered infections, tend to be short-lived after infection with the closely related coronavirus SARS-CoV, which causes severe acute respiratory syndrome (SARS).

In contrast, another type of immune cell called a memory T cell, which can recognize a previously encountered pathogen and initiate an immune response to it, may persist for years after the initial infection.

In a new study, memory T cells protected against SARS-CoV-2 infection, even in the absence of antibodies against the virus.
The new research features in the journal Cell.

Five categories of exposure

In the new research, Buggert and his colleagues investigated the immune status of 206 individuals in Sweden, where measures to control the spread of SARS-CoV-2 have been less strict than in other European countries.

Their participants fell into five categories:

• people with ongoing moderate or severe COVID-19
• individuals convalescing after a mild or severe infection
• asymptomatic family members exposed to the infection
• healthy individuals who donated blood during the pandemic
• healthy individuals who donated blood in 2019, before the pandemic


As expected, the team found strong memory T-cell responses and high levels of antibodies specific to the virus in all 23 people who had recovered from severe COVID-19.

More surprisingly, 30 of the 31 people who recovered from a mild infection had memory T-cell responses to the virus, and 27 had antibodies against it.

Out of 28 family members exposed to an infected individual, 26 were able to mount T-cell responses to the virus, and 17 had antibodies against it.

Even after a very mild infection, memory T-cell responses were often detectable months later, sometimes even in the absence of SARS-CoV-2 antibodies.

“Our findings suggest that the reliance on antibody responses may underestimate the extent of population-level immunity against SARS-CoV-2,” says Buggert, the senior author of the study. “The obvious next step is to determine whether robust memory T-cell responses in the absence of detectable antibodies can protect against COVID-19 in the long term.”

“Our collective dataset shows that SARS-CoV-2 elicits robust, broad, and highly functional memory T-cell responses, suggesting that natural exposure or infection may prevent recurrent episodes of severe COVID-19.”
– The study authors

General coronavirus immunity
Remarkably, in 28% of those who had donated blood samples in 2019, before the current pandemic, the researchers detected T cells that reacted to SARS-CoV-2. The researchers believe that this reflects immunity induced by exposure to other coronaviruses that have protein sequences in common with SARS-CoV-2.

They speculate that these responses may provide some protection against the new coronavirus, though direct evidence for this is currently lacking.

The authors acknowledge that their study was limited by the small numbers in each group and lack of clinical follow-up. “It, therefore, remains to be determined if robust memory T-cell responses in the absence of detectable circulating antibodies can protect against severe forms of COVID-19,” they write.

Thus far, however, none of the individuals in their study who recovered from the infection have experienced further episodes of COVID-19.

In addition, the authors cite research showing that rhesus macaques infected with SARS-CoV-2 develop almost complete immunity against future infections with the virus.

There are also no confirmed cases of humans who have had COVID-19 contracting the infection again at a later date.



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2020-08-24T11:04:46Z
Dr. Risch on HCQ: https://youtu.be/lV-KeieI2s8 

Kill Shot at 3:35....[u
Quote:

Dr. Fauci and the FDA re doing the same thing that was done in 1987, and this has lead to the deaths of hundreds of thousands of people that could have been saved by this drug.

[/u]

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Zero2Cool
2020-08-24T13:19:28Z
This makes it look like things might be getting gooder.
 image.png You have insufficient rights to see the content.

Basically, if you're 44 years of age or younger, there's less than 1% chance of COVID-19 being the cause of your death. This is insane how strongly affects people in the second half of their life. My goodness.
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Cheesey
2020-08-24T14:55:04Z
Originally Posted by: Zero2Cool 

This makes it look like things might be getting gooder.
 image.png You have insufficient rights to see the content.

Basically, if you're 44 years of age or younger, there's less than 1% chance of COVID-19 being the cause of your death. This is insane how strongly affects people in the second half of their life. My goodness.
 image.png You have insufficient rights to see the content.



My take on this is that a lot of people over age 44 also aren’t as active physically. Thus more likely to get sick. Just look around you and see how many older people are horribly out of shape.
I’m 63 years old, but try to walk a lot to keep healthy. Since my open heart surgery in 2009, I went from 220 pounds down to what I am now at 163. And I’ve kept it off. I’m sure that has helped my over all health.
Of course that doesn’t mean I won’t catch the disease. But maybe if I do I have a better chance as an old guy to be able to survive.
At least I hope that’s the case. It’s the only my thoughts on it.
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KRK
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2020-08-24T16:01:50Z
In the not so great news department.....
https://www.medpagetoday...08-24&eun=g430804d0r 

This has negative ramifications for vaccines as well.....a great therapeutic is typically better than vaccine. I just wish the silver bullet existed.
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Cheesey
2020-08-24T18:04:15Z
Originally Posted by: KRK 

. I just wish the silver bullet existed.



It does.....but it would only work if you wanted to kill a werewolf that had covid.
😜
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2020-08-24T22:41:23Z
Worth the 10 minute listen especially if you or someone you love is high risk
https://twitter.com/i/status/1297715098795094017 

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Zero2Cool (25-Feb) : #Packers expected to have interest in Lavonte David per @CBSSports #NFL #Greenbay
beast (25-Feb) : On one hand, I think this is sounding more like the Packers have a shot at signing Watt, on the other, if he was, why wouldn't he of signed with his childhood team with best QB already?
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Zero2Cool (20-Feb) : @nflrums #Steelers expected to release CB Joe Haden saving the team 7M in cap space
Zero2Cool (19-Feb) : Sources say Wagner is contemplating retirement. The moves clear $10.25M in cap space.
Zero2Cool (18-Feb) : #Packers likely to part ways with Josh Jackson CB #Iowa #GreenBay #NFL
Zero2Cool (18-Feb) : Philadelphia has agreed to trade Carson Wentz to the Indianapolis Colts in exchange for a 2021 third-round pick and a conditional 2022 second-round pick that could turn into a first, league sources te
Zero2Cool (18-Feb) : The NFL in a memo to teams has said the salary cap floor has been increased from $175 million to $180 million after talks with the NFLPA.
Zero2Cool (17-Feb) : Richard Sherman: It’s been made pretty clear I won’t return to 49ers
Zero2Cool (17-Feb) : Graziano: #Broncos are “likely” to decline Von Miller’s $18M team option. That would make him an unrestricted free agent.
Mucky Tundra (16-Feb) : Thems are fighting words down South Zero! Well, when the snow and ice melts and they run out of bread and milk
Zero2Cool (15-Feb) : WINTER STORM WARNING... Yes. Bring it biotches
Zero2Cool (14-Feb) : “Why go from $175 million for 2021 and then raise it to $220 million in 2022? That just doesn’t make any sense,” one NFL agent he said.
Zero2Cool (13-Feb) : @ProFootballTalk Report: Steelers, Browns, Titans, Bills have shown interest in J.J. Watt
Zero2Cool (12-Feb) : JJ Watt asks for and is released by Texans. I bet he joins his brother.
packerfanoutwest (12-Feb) : Isn't that the title Capers has in Detroit?
Zero2Cool (10-Feb) : Report: Mike Pettine joining Bears as senior defensive assistant
Zero2Cool (10-Feb) : @MikeKlis Broncos are releasing CB A.J. Bouye per source. #9sports
Cheesey (10-Feb) : Yup, Mucky. Brady is “the chosen one”. He could knife a guy in the field, and they’d flag the other guy for bleeding!
Mucky Tundra (10-Feb) : Zero, I wonder if Wisconsin promised him the HC at some point in the future
Mucky Tundra (10-Feb) : yeah Cheesey,I love how Brady ran 10+ yards and bumps into Mathieu helmet first the refs do nothing but when Mathieu turns to walk away and just points at Brady they instantly flag him
Zero2Cool (10-Feb) : Oates: Leonhard "dang near took the job"
Zero2Cool (10-Feb) : Tell your friend registration is free. Register and ask.
packerfanoutwest (10-Feb) : Was the SB fixed? asking for a friend
Cheesey (10-Feb) : Easy to be a “tough guy” when your defense is killing the other team.
Zero2Cool (9-Feb) : I'm serious. Dude wants to crush people and apologize later. Kill!!!
Cheesey (9-Feb) : Yup Mucky! I agree! A killer D can make any QB look good.
Mucky Tundra (9-Feb) : Cheesey, Trent Dilfer could win a super with that team and probably had a higher completion percentage against Wash and NO and no floating passes
Mucky Tundra (9-Feb) : Zero, I cant tell if you're being sarcastic, serious or both lol
Cheesey (9-Feb) : If Rodgers has Tampa’s defense, he would win SBs too.
Zero2Cool (9-Feb) : I believe that. I love the guys fire and killer instinct. Wish we had a QB with that.
Mucky Tundra (9-Feb) : Zero, Brady just wants it more than the next guy
Zero2Cool (9-Feb) : Former NFL coach Marty Schottenheimer has passed away,
Zero2Cool (9-Feb) : The defense is because of Brady. That's how amazing great he is.
Mucky Tundra (9-Feb) : The Patriots and Bucs D allowed 0 TDs in their last two super bowl victories. unreal
Mucky Tundra (9-Feb) : and it just looked a little funky to anyone since they took away stops from the Chiefs D and Tampa scored TDs off the drives
Mucky Tundra (9-Feb) : Zero, the penalties were a little iffy and compared to what was going on when GB played Tampa absolutely nothing. Throw in the timing of them (bailed Tampa out of INT and a failed 3rd down conversion)
Zero2Cool (8-Feb) : The #Cardinals and long-time Pro Bowl CB Patrick Peterson are parting ways, per @MattVerderame
Zero2Cool (8-Feb) : It was funny reading comments. Brady is amazing! I look at scoreboard, 9 points? Brady did that? lol
Zero2Cool (8-Feb) : I remembered it and started following in Twitter. Seems the officials were thinking the game was bout them
yooperfan (8-Feb) : Just another fixed game by the NFL. The officiating decided another game. I’m sick of it.🤬
Cheesey (8-Feb) : Mahomes has to run for his life all game.
Mucky Tundra (8-Feb) : Zero, I was watching it closely until the Fournette TD in the 3rd. Was hoping Mahomes would put on a show
Cheesey (8-Feb) : I didn’t care who won. Besides, I don’t have “Hulu”! 😂
packerfanoutwest (8-Feb) : Fuck Brady and the refs
Zero2Cool (8-Feb) : No shame in it. Just have no interest in it. Deadliest Decade 90s got me all day.
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